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C1q腫瘤壞死因子相關(guān)蛋白3(C1QTNF3)單克隆抗體

Monoclonal Antibody to C1q And Tumor Necrosis Factor Related Protein 3 (C1QTNF3)

CORS26; CTRP3; Corcs; Cors; Cartonectin; Collagenous repeat-containing sequence 26 kDa protein

  • C1q腫瘤壞死因子相關(guān)蛋白3(C1QTNF3)單克隆抗體產(chǎn)品包裝(模擬)
  • C1q腫瘤壞死因子相關(guān)蛋白3(C1QTNF3)單克隆抗體產(chǎn)品包裝(模擬)
  • Certificate通過(guò)ISO 9001、ISO 13485質(zhì)量體系認(rèn)證

特異性

該抗體是針對(duì)C1QTNF3的小鼠單克隆抗體。在免疫組織化學(xué)染色和免疫印跡實(shí)驗(yàn)中能識(shí)別C1QTNF3。

用法

免疫印跡：0.2-2μg/mL;1:500-5000
免疫組織化學(xué)：5-20μg/mL;1:50-200
免疫細(xì)胞化學(xué)：5-20μg/mL;1:50-200
最佳稀釋倍數(shù)最終由用戶決定。

儲(chǔ)存

經(jīng)常使用則4°C保存。-20°C保存不超過(guò)兩年。避免反復(fù)凍融。

穩(wěn)定性

熱穩(wěn)定性以損失率顯示。損失率是由加速降解試驗(yàn)決定，具體方法如下：在37°C孵育48小時(shí)，沒(méi)有顯著的降解或者沉淀產(chǎn)生。保質(zhì)期內(nèi)，在適當(dāng)?shù)臈l件下存儲(chǔ)，損失率低于5%。

贈(zèng)品

相關(guān)產(chǎn)品

編號(hào)適用物種：Homo sapiens (Human，人)應(yīng)用(僅供研究使用，不用于臨床診斷！)
RPK169Hu01C1q腫瘤壞死因子相關(guān)蛋白3(C1QTNF3)重組蛋白Positive Control; Immunogen; SDS-PAGE; WB.
PAK169Hu01C1q腫瘤壞死因子相關(guān)蛋白3(C1QTNF3)多克隆抗體WB; IHC; ICC; IP.
MAK169Hu21C1q腫瘤壞死因子相關(guān)蛋白3(C1QTNF3)單克隆抗體WB; IHC; ICC; IP.
SEK169HuC1q腫瘤壞死因子相關(guān)蛋白3(C1QTNF3)檢測(cè)試劑盒(酶聯(lián)免疫吸附試驗(yàn)法)Enzyme-linked immunosorbent assay for Antigen Detection.
LMK169HuC1q腫瘤壞死因子相關(guān)蛋白3(C1QTNF3)等多因子檢測(cè)試劑盒(流式熒光發(fā)光法)FLIA Kit for Antigen Detection.

參考文獻(xiàn)

雜志參考文獻(xiàn)
PLoS One. Low Serum Cartonectin/CTRP3 Concentrations in Newly Diagnosed Type 2 Diabetes Mellitus: In Vivo Regulation of Cartonectin by Glucose[Pubmed:25409499]
BMC Cardiovascular DisordersSerum levels of C1q/TNF-related protein-1 (CTRP-1) are closely associated with coronary artery disease[Pubmed:27169633]
Journal of?Diabetes?ResearchPlasma C1q/TNF-Related Protein-3 (CTRP-3) and High-Mobility Group Box-1 (HMGB-1) Concentrations in Subjects with Prediabetes and Type 2 Diabetes[pubmed:27738641]
Journal of Clinical MedicineCirculating C1q/TNF-Related Protein 3, Omentin-1 and NGAL in Obese Patients with Type 2 Diabetes During Insulin Therapy[Pubmed: 31195747]
Cell Death & DiseaseC1q-TNF-related protein-3 attenuates pressure overload-induced cardiac hypertrophy by suppressing the p38/CREB pathway and p38-induced ER stress[Pubmed: 31285424]